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negative control igg chip-it® control qpcr kit – human  (Active Motif)


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    Active Motif negative control igg chip-it® control qpcr kit – human
    Negative Control Igg Chip It® Control Qpcr Kit – Human, supplied by Active Motif, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/negative+control+igg+kit/pmc08694049__pnas__2111643118__sapp-57-52-63?v=Active+Motif
    Average 90 stars, based on 1 article reviews
    negative control igg chip-it® control qpcr kit – human - by Bioz Stars, 2026-07
    90/100 stars

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    (A) Principal-component analysis (PCA) <t>of</t> <t>CUT&Tag</t> data shows a shift in the active transcription mark H3K4me3 in KT-47PT (orange, right; n = 2 biologically independent samples) compared to KT-47R (pink, left; n = 3) along principal component 1 (PC1, 78.9% variance). (B) H3K4me3 CUT&Tag Gene Cloud highlights increased H3K4me3 at HOXB and C clusters and nephrogenic stem cell gene promoters. (C) Heatmap displays the top 30 gene promoters with H3K4me3 enrichment in KT-47R versus KT-47PT. (D) PCA of CUT&Tag data shows a shift in the repressive transcription mark H3K27me3 in KT-47PT (red, right; n = 3) compared to KT-47R (blue, left; n = 3) along PC1 (78.9% variance). (E) H3K27me3 CUT&Tag Gene Cloud indicates decreased H3K27me3 at HOXB and nephrogenic stem cell genes. (F) Heatmap displays the top 30 gene promoters with H3K27me3 enrichment in KT-47R versus KT-47PT. (G) PCA and (H) unsupervised hierarchical clustering of methylation EPIC data demonstrate that the predominant shift in DNA methylation occurs during the development of chemotherapy resistance ( n = 16 biologically independent samples).
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    (A) Principal-component analysis (PCA) <t>of</t> <t>CUT&Tag</t> data shows a shift in the active transcription mark H3K4me3 in KT-47PT (orange, right; n = 2 biologically independent samples) compared to KT-47R (pink, left; n = 3) along principal component 1 (PC1, 78.9% variance). (B) H3K4me3 CUT&Tag Gene Cloud highlights increased H3K4me3 at HOXB and C clusters and nephrogenic stem cell gene promoters. (C) Heatmap displays the top 30 gene promoters with H3K4me3 enrichment in KT-47R versus KT-47PT. (D) PCA of CUT&Tag data shows a shift in the repressive transcription mark H3K27me3 in KT-47PT (red, right; n = 3) compared to KT-47R (blue, left; n = 3) along PC1 (78.9% variance). (E) H3K27me3 CUT&Tag Gene Cloud indicates decreased H3K27me3 at HOXB and nephrogenic stem cell genes. (F) Heatmap displays the top 30 gene promoters with H3K27me3 enrichment in KT-47R versus KT-47PT. (G) PCA and (H) unsupervised hierarchical clustering of methylation EPIC data demonstrate that the predominant shift in DNA methylation occurs during the development of chemotherapy resistance ( n = 16 biologically independent samples).
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    (A) Principal-component analysis (PCA) <t>of</t> <t>CUT&Tag</t> data shows a shift in the active transcription mark H3K4me3 in KT-47PT (orange, right; n = 2 biologically independent samples) compared to KT-47R (pink, left; n = 3) along principal component 1 (PC1, 78.9% variance). (B) H3K4me3 CUT&Tag Gene Cloud highlights increased H3K4me3 at HOXB and C clusters and nephrogenic stem cell gene promoters. (C) Heatmap displays the top 30 gene promoters with H3K4me3 enrichment in KT-47R versus KT-47PT. (D) PCA of CUT&Tag data shows a shift in the repressive transcription mark H3K27me3 in KT-47PT (red, right; n = 3) compared to KT-47R (blue, left; n = 3) along PC1 (78.9% variance). (E) H3K27me3 CUT&Tag Gene Cloud indicates decreased H3K27me3 at HOXB and nephrogenic stem cell genes. (F) Heatmap displays the top 30 gene promoters with H3K27me3 enrichment in KT-47R versus KT-47PT. (G) PCA and (H) unsupervised hierarchical clustering of methylation EPIC data demonstrate that the predominant shift in DNA methylation occurs during the development of chemotherapy resistance ( n = 16 biologically independent samples).
    Negative Control Igg Chip It® Control Qpcr Kit – Human, supplied by Active Motif, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 90 stars, based on 1 article reviews
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    1 ml of rabbit igg from the kit as a negative control  (DIAGENODE DIAGNOSTICS)
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    DIAGENODE DIAGNOSTICS 1 ml of rabbit igg from the kit as a negative control
    (A) Principal-component analysis (PCA) <t>of</t> <t>CUT&Tag</t> data shows a shift in the active transcription mark H3K4me3 in KT-47PT (orange, right; n = 2 biologically independent samples) compared to KT-47R (pink, left; n = 3) along principal component 1 (PC1, 78.9% variance). (B) H3K4me3 CUT&Tag Gene Cloud highlights increased H3K4me3 at HOXB and C clusters and nephrogenic stem cell gene promoters. (C) Heatmap displays the top 30 gene promoters with H3K4me3 enrichment in KT-47R versus KT-47PT. (D) PCA of CUT&Tag data shows a shift in the repressive transcription mark H3K27me3 in KT-47PT (red, right; n = 3) compared to KT-47R (blue, left; n = 3) along PC1 (78.9% variance). (E) H3K27me3 CUT&Tag Gene Cloud indicates decreased H3K27me3 at HOXB and nephrogenic stem cell genes. (F) Heatmap displays the top 30 gene promoters with H3K27me3 enrichment in KT-47R versus KT-47PT. (G) PCA and (H) unsupervised hierarchical clustering of methylation EPIC data demonstrate that the predominant shift in DNA methylation occurs during the development of chemotherapy resistance ( n = 16 biologically independent samples).
    1 Ml Of Rabbit Igg From The Kit As A Negative Control, supplied by DIAGENODE DIAGNOSTICS, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/negative+control+igg+kit/pm32234487-321-29-12?v=DIAGENODE+DIAGNOSTICS
    Average 90 stars, based on 1 article reviews
    1 ml of rabbit igg from the kit as a negative control - by Bioz Stars, 2026-07
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    Image Search Results


    (A) Principal-component analysis (PCA) of CUT&Tag data shows a shift in the active transcription mark H3K4me3 in KT-47PT (orange, right; n = 2 biologically independent samples) compared to KT-47R (pink, left; n = 3) along principal component 1 (PC1, 78.9% variance). (B) H3K4me3 CUT&Tag Gene Cloud highlights increased H3K4me3 at HOXB and C clusters and nephrogenic stem cell gene promoters. (C) Heatmap displays the top 30 gene promoters with H3K4me3 enrichment in KT-47R versus KT-47PT. (D) PCA of CUT&Tag data shows a shift in the repressive transcription mark H3K27me3 in KT-47PT (red, right; n = 3) compared to KT-47R (blue, left; n = 3) along PC1 (78.9% variance). (E) H3K27me3 CUT&Tag Gene Cloud indicates decreased H3K27me3 at HOXB and nephrogenic stem cell genes. (F) Heatmap displays the top 30 gene promoters with H3K27me3 enrichment in KT-47R versus KT-47PT. (G) PCA and (H) unsupervised hierarchical clustering of methylation EPIC data demonstrate that the predominant shift in DNA methylation occurs during the development of chemotherapy resistance ( n = 16 biologically independent samples).

    Journal: Cell reports

    Article Title: Developmental reprogramming underlies chemotherapy resistance in favorable-histology Wilms tumor

    doi: 10.1016/j.celrep.2026.117063

    Figure Lengend Snippet: (A) Principal-component analysis (PCA) of CUT&Tag data shows a shift in the active transcription mark H3K4me3 in KT-47PT (orange, right; n = 2 biologically independent samples) compared to KT-47R (pink, left; n = 3) along principal component 1 (PC1, 78.9% variance). (B) H3K4me3 CUT&Tag Gene Cloud highlights increased H3K4me3 at HOXB and C clusters and nephrogenic stem cell gene promoters. (C) Heatmap displays the top 30 gene promoters with H3K4me3 enrichment in KT-47R versus KT-47PT. (D) PCA of CUT&Tag data shows a shift in the repressive transcription mark H3K27me3 in KT-47PT (red, right; n = 3) compared to KT-47R (blue, left; n = 3) along PC1 (78.9% variance). (E) H3K27me3 CUT&Tag Gene Cloud indicates decreased H3K27me3 at HOXB and nephrogenic stem cell genes. (F) Heatmap displays the top 30 gene promoters with H3K27me3 enrichment in KT-47R versus KT-47PT. (G) PCA and (H) unsupervised hierarchical clustering of methylation EPIC data demonstrate that the predominant shift in DNA methylation occurs during the development of chemotherapy resistance ( n = 16 biologically independent samples).

    Article Snippet: Rabbit IgG negative control antibody (CUT&Tag) , EpiCypher , cat#13–0042; RRID:AB_2923178.

    Techniques: Methylation, DNA Methylation Assay

    (A) Genome-wide matrix of topologically associated domains (TADs) shows that most interactions are intrachromosomal (diagonal intensity pattern indicates each chromosome plotted against itself). A unique interaction pattern between chromosomes 1p and 7q (black arrow) was observed in KT-47R1 but not in KT-47PT. (B) Hi-C identified an interchromosomal interaction between chromosome 1p32.3 ( GLIS1 locus) and chromosome 7q21 ( ABCB1 locus, black arrow) in KT-47R1. The light red vertical line marks the boundary between interacting regions of chromosomes 1 (right) and 7 (left). Red arcs indicate interactions between chromosomes 1 and 7 in KT-47R1, absent in KT-47PT (no blue arcs crossing the boundary). RNA-seq reads confirm upregulation of ABCB1 (black arrow) in KT-47R1. (C) CUT&Tag analysis shows H3K4me1 peaks within the interacting region of chromosome 1p32.3. Peaks are present in both KT-47PT and KT-47R1, but the chromatin interaction occurs only in KT-47R1. (D) FISH 1p32.3 break-apart analysis demonstrates no translocation involving 1p32.3 in KT-47PT, KT-47 pre-resistance, KT-47R1, or KT-47R2. Scale bars, 5 μm. (E) Chromosome 1 and 7 painting analysis indicated increased interaction (colocalization) between chromosomes 1 and 7 in KT-47R1 (48%) and KT-47R2 (48%) when compared to KT-47PT (19%) and KT-47PR (25%). Scale bars, 10 μm. n = 2 samples per group.

    Journal: Cell reports

    Article Title: Developmental reprogramming underlies chemotherapy resistance in favorable-histology Wilms tumor

    doi: 10.1016/j.celrep.2026.117063

    Figure Lengend Snippet: (A) Genome-wide matrix of topologically associated domains (TADs) shows that most interactions are intrachromosomal (diagonal intensity pattern indicates each chromosome plotted against itself). A unique interaction pattern between chromosomes 1p and 7q (black arrow) was observed in KT-47R1 but not in KT-47PT. (B) Hi-C identified an interchromosomal interaction between chromosome 1p32.3 ( GLIS1 locus) and chromosome 7q21 ( ABCB1 locus, black arrow) in KT-47R1. The light red vertical line marks the boundary between interacting regions of chromosomes 1 (right) and 7 (left). Red arcs indicate interactions between chromosomes 1 and 7 in KT-47R1, absent in KT-47PT (no blue arcs crossing the boundary). RNA-seq reads confirm upregulation of ABCB1 (black arrow) in KT-47R1. (C) CUT&Tag analysis shows H3K4me1 peaks within the interacting region of chromosome 1p32.3. Peaks are present in both KT-47PT and KT-47R1, but the chromatin interaction occurs only in KT-47R1. (D) FISH 1p32.3 break-apart analysis demonstrates no translocation involving 1p32.3 in KT-47PT, KT-47 pre-resistance, KT-47R1, or KT-47R2. Scale bars, 5 μm. (E) Chromosome 1 and 7 painting analysis indicated increased interaction (colocalization) between chromosomes 1 and 7 in KT-47R1 (48%) and KT-47R2 (48%) when compared to KT-47PT (19%) and KT-47PR (25%). Scale bars, 10 μm. n = 2 samples per group.

    Article Snippet: Rabbit IgG negative control antibody (CUT&Tag) , EpiCypher , cat#13–0042; RRID:AB_2923178.

    Techniques: Genome Wide, Hi-C, RNA Sequencing, Translocation Assay